Hydrogen has long been considered inconspicuous: colorless, odorless, and chemically very stable. But new scientific findings show that this smallest of all atoms, existing as a molecule, performs surprisingly active tasks in the human body. Its effect in the mitochondria—the tiny “powerhouses” of our cells responsible for energy production—is particularly fascinating.
Recent research shows that, in addition to its previously known property of neutralizing harmful substances such as free radicals/oxygen radicals, hydrogen also specifically intervenes in other processes. Hydrogen influences a key protein in energy production, the Rieske iron-sulfur protein, and thus temporarily slows down energy production. This might initially sound like a disadvantage, but it is actually a clever protective mechanism. The cell uses this brief pause to essentially clean up, carry out repairs, and make itself more resilient.
Hydrogen for cell protection and regeneration
Hydrogen doesn’t act like a conventional drug that suppresses symptoms. Instead, it sends a signal to the cell to self-regulate. This gentle, regulatory effect could explain why hydrogen has been linked to positive effects on well-being, cell protection, and regeneration in various studies.
This new perspective makes it clear: hydrogen is not a “miracle cure,” but an intelligent catalyst for the cell. Its strength lies in activating the body’s own protective and repair mechanisms. As a result, hydrogen is increasingly becoming the focus of modern research as a supportive companion for health and cellular balance.
The research was conducted by:
- Department of Neurogenetics, Center for Neurological Diseases and Cancer, Nagoya University School of Medicine, Japan
- Department of Materials Science (Physics), Graduate School of Natural Sciences, Nagoya University, Japan
- Department of Kinesiology and Outdoor Recreation, Southern Utah University, Cedar City, USA
- Institute of Molecular Hydrogen, Enoch, USA
- Graduate School of Nutritional Sciences, Nagoya University of Arts and Sciences, Japan
The research was supported by:
- Research grant from the Japan Agency for Medical Research and Development
- Japan Society for the Advancement of Science
- Japan Ministry of Health, Labour and Welfare
- National Center for Neurology and Psychiatry
Quelle doi.org/10.1016/j.redox.2025.103952
Further studies and research findings
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